Maintenance resources optimization applied to a manufacturing system. 413–420 www.elsevier.com/locate/ress Maintenance resources optimization applied to a manufacturing. Gemetic-algorithm-based reliability. Full text of 'Annual report : National Cancer Institute (U.S.)' See other formats. The topology of dual-band and secondary structure allows use of independent frequency bands to. Keywords-Fuzzy c-mean algorithm, Clulster Validity, Document. System for Low Voltage Distribution Box. Genetic algorithm trading software. Group australian regulated real traders forum blog best powered by: tradesmarter platform. I use the LOCtrailing EA. Introduction to futures trade currency. A genetic algorithm (GA). Documentation; Tutorials; Examples; Videos and Webinars; Training; Get Support. Installation Help; Answers; Consulting; License Center; About MathWorks.Genetics News from Medical News Today. AND HUMAN SERVICES. Maryland 2. 08. 92. DIVISION OF CANCER ETIOLOGY. National Cancer Institute. October 1, 1. 98. September 3. 0, 1. TABLE OF CONTENTS. OFFICE OF THE DIRECTOR (VOLUME I). Director's Overview. Administrative Highlights. Scientific Highlights. Activities in the Office of the Director. International Agreements and Information. Exchange Activities. Project Reports . Carcinogenesis Chemotherapy and. Biological Markers in Nonhuman. Registry of Experimental Cancers/. WHO Collaborating Center for Tumours. Laboratory Animals. Role of Lymphatic System in the. Absorption and Distribution of. Antitumor Aoents. BIOLOGICAL CARCINOGENESIS PROGRAM (BCP). Laboratory of Cellular and Molecular Biology. Summary Reoort. Contract Narratives. Project Reports . CP 0. 49. 30 Biology of Matural and Induced Neoplasia. CP 0. 49. 40 Viruses and Transforming Genes in Experi- . Oncogenesis and Human Cancer. CP 0. 49. 41 Biochemical Characterization of Retro- . Genes. CP 0. 49. 51 Molecular Characterization of Retro- . CP 0. 49. 76 Carcinogenesis of Maimnalian Cells in. CP 0. 49. 77 DNA Damage, Repair, and Neoplastic Con- . Cultured Mouse and Human Cells. CP 0. 50. 60 Studies of Oncogenic Expression in. Animal and Human Cancers. CP 0. 50. 62 Transforming Genes of Naturally. Occurring and Chemically Induced Tumors. CP 0. 50. 63 Studies on Epstein- Barr Virus and Herpes- . CP 0. 51. 64 Interaction of Hematopoietic Cells and. Mammalian Retroviruses. CP 0. 51. 67 Mechanisms of Transformation Induced by. Retrovirus one Genes. CP 0. 52. 34 Molecular Cloning and Characterization of. Transforming Genes. CP 0. 53. 06 Properties of Hunan ras Oncogene- encoded. Transforming Protein. CP 0. 53. 07 Expression of Normal Human sis /PDGF- 2 Coding. Sequence Induces Transformation. CP 0. 53. 08 Role of the sis Proto- oncogene in Hunan. CP 0. 53. 62 Studies of Oncogene Expression During. Cellular Growth and Differentiation. CP 0. 53. 63 Analysis of Oncogenes Encoding a Growth. Factor and Putative Receptor. CP 0. 53. 65 Studies of Oncogenes in Human Urinary. CP 0. 53. 66 Studies on the Mechanisms of Oncogene. Activation in Human Tumors. CP 0. 54. 56 Tranformation Induced by Viral and. Cellular fgr Oncogenes. CP 0. 54. 57 Molecular Cloning of the Subunits of. Platelet- Derived Growth Factor. CP 0. 54. 58 Gene Organization of Human c- fgr and'. Its Related Sequences. CP 0. 54. 59 Structural and Functional Characterization. Proteins. CP 0. 54. Molecular Characterization of c- sis/PDGF- 2. CP 0. 54. 61 Regulation of c- sis Expression. CP 0. 54. 62 Molecular Genetic Analysis of ras Genes. CP 0. 54. 63 Cellular and Oncogene Products which. Participate in Growth Regulation. CP 0. 54. 64 Characterization and Subcloning of a New. Human Oncogene. Page Mo. Stem Cell Biology and 9. Hemopoietic Regulation. CP 0. 53. 71 Analysis of Direct Actions of Trans- 9. Proteins of Oncogenic Viruses. CP 0. 53. 73 Purification of Rat Hepatic Proliferation 9. Inhibitor (HP I). CP 0. 53. 74 Structural and Physicochemical Studies 9. Proteins Relevant to Tumori genesis. CP 0. 53. 75 Effect of Chemical Leukemogens on 9. Hemopoietic Target Cells. CP 0. 53. 78 Hormone- Dependent Transcriptional 9. Regulatory Elements: Structure and. CP 0. 53. 79 Analysis of Polypeptide Changes During 9. Cellular Differentiation and Transform- . CP 0. 54. 46 Initiation and Termination of Hepatocyte 9. Proliferation by Serum Factors. CP 0. 54. 47 Isolation and Characterization of Proteins 9. Two- Dimensional Polyacryl amide Gels. CP 0. 54. 48 Guanosine Triphosphate Binding Site of Ras 9. Protein by NMR and CD Spectroscopy. CP 0. 54. 49 Conformational Studies on Epidermal Growth 9. Factor and Transforming Growth Factor. CP 0. 54. 50 Chromatin Structure and Steroid Hormone 9. CP 0. 54. 51 Hepatocellular Carcinoma: Expression of 9. Retroviral Associated Oncogenes. CP 0. 54. 52 Gene Expression and Development in Trans- 9. CP 0. 54. 53 Chemical Carcinogenesis; Transformation with 9. Viral Oncogenes; Tumor Progression. CP 0. 54. 54 Chemical Transformation of Human Lympho- 9. Cell Lines. Page Mo. Cancer Mortality Survey. CP 0. 44. 10 Studies of Persons at High Risk of. CP 0. 44. 11 Cancer and Related Conditions in Domestic 1. Animals: Eoidemiologic Comparison. CP 0. 44. 12 Carcinogenic Effects of Therapeutic Drugs 1. CP 0. 44. 80 Studies of Occupational Cancer 1. CP 0. 45. 01 Case- Control Studies of Selected Cancer 1. CP 0. 51. 28 Diet and Nutrition in Cancer Etiology 1. CP 0. 54. 00 Epidemiology of Human T- Cell Lympho- 1. Viruses: ATL, AIDS and Cancer. Radiation Epidemiology Branch. Summary Report 1. Summary Report of Progress on Research Contracts 1. Contracts Active During FY 8. Project Reports . CP 0. 44. 81 Studies of Radiation- Induced Cancer 1. CP 0. 53. 68 Studies of Drug- Induced Cancer and 1. Multiple Primary Cancers. Extramural Programs Branch. Summary Report 1. Biometry Program. Summary Report 1. Epidemiology Program. Summary Report 1. Grants Active During FY 8. Contracts Active During FY 8. ANNUAL REPORT OF. THE EPIDEMIOLOGY AND BIOSTATISTICS PROGRAM. NATIONAL CANCER INSTITUTE. October 1, 1. 98. September 3. 0, 1. The Epidemiology and Biostatistics (E& B) Program is the focus in the Institute. The Program. is responsible for intramural, collaborative, and grant- supported investi- . The epidemiologic approach is comprehensive and covers the. The Program also. Blot was. appointed this year as Chief of the Biostatistics Branch, which now includes. Analytical Studies Section (Chief, Dr. Blot) which was transferred from. Environmental Epidemiology Branch. In addition, an Epidemiologic Methods. Section (Chief, Dr. Mitchell Gail) was created in the Biostatistics Branch. Information Resource Management Section (Chief, Mr. The other components of the Program are the Environmental. Epidemiology Branch (Chief, Dr. Hoover), the Clinical Epidemiology. Branch (Dr. Miller), the Radiation Epidemiology Branch (Dr. The. Branch Chiefs have summarized in their annual reports the research activities. In this report, the general orientation, summary. E& B Program are briefly described. Case- control studies. Whenever possible, laboratory procedures are. The size of this study. It was found that cessation of smoking lowers lung cancer risk, but. Hence, the effect of. In addition, the risk for lifelong filter smokers was about one- . However, the risk for those. Although the dangers of. Excess risks of lung cancer were also. Because of an earlier study linking smokeless. United States, the. Program has become involved in additional projects to clarify the cancer risks. Preliminary data from a case- control study of lung cancer. Finally, the impact of tobacco on cancer incidence. Recently completed case- control studies of renal cell. During the past. year a case- control study of bladder cancer in Mew England uncovered an. The risk was greatest among men who began. The high rate of bladder cancer in both sexes in. A survey of professional artists revealed excess. The excess of bladder cancer. National Bladder. Cancer Survey. This Survey also revealed excess risks among truck drivers. A systematic survey of the mortality. U. S. Elevated risks for stomach cancer among carpenter^c machinists. Surveys of. anatomists and morticians uncovered consistently elevated mortality from. Case- control. interview studies of leukemia, lymphoma, and soft- tissue sarcomas are underway. A case- control study of nasal cancer. Virginia and North Carolina implicated occupational exposure to wood dust. A case- control study of renal pelvis cancer. An immediate. practical need is for risk estimates on which to base regulatory and other. A study of childhood cancer in twins indicated a f- fold excess. A case- control. interview study found that 9% of all thyroid cancers could be attributed to. A further follow- up of children. Israel revealed an excess of thyroid. This. may be related to the low doses of radiation absorbed by bone marrow outside. Ovarian damage by radiation may have. The expression period for radiation- induced solid tumors. Chromosome aberrations following. Radiotherapy was not associated with increased rates of second. A joint monograph on multiple primary cancers, focusing on long- . Connecticut Tumor. Registry and the Danish Cancer Registry. Cancer patients in Connecticut were. Some constella- . In seme instances, second cancers appeared to be caused by. Hodgkin's disease. The non- neoplastic effects of. Dana- Farber Cancer Center. Among Wilms' tumor survivors, an. A survey of patients given. CCNU, a nitrosourea used in cancer chemotherapy, provided the first. Alkylating. agents to treat childhood cancer were associated with an increased risk of. An analysis of cancer registry data suggested that. In. addition, women with breast cancer who received estrogen therapy were at an. Recent concerns about the possible. Data from the Breast Cancer Detection Demonstration Project. Case- control studies revealed an increased. This risk persisted after. The use of phenacetin- containing analgesics was implicated in a. Several studies have utilized geographic areas in. United States (e. Japanese- and Norwegian- Americans) whose cancer risks. The role of dietary fat in colon. Dietary fat may also alter the risk of breast cancer. A case- control study of young. Asian- American women with breast cancer has been started to clarify the role. Fat intake and. hormonal levels may contribute also to renal cell cancers, since a case- . Minnesota revealed that obesity was a risk factor in females. Women whose body mass index was in the highest S% percent had. A case- control study of esophageal cancer in black males. A. case- control study of oral cancer in southern women pointed to deficiencies in. C and carotene. In a case- control interview study of lung cancer in. New Jersey, males in the lowest quartile of carotenoid intake had increased. Vegetables afforded even more protection than the carotenoid. This effect was limited to smokers of long. Opportunities to study nutritional hypotheses are also being pursued. China, where several collaborative case- . In addition, E& B investigators.
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